Current HIV Research covers all the latest and outstanding developments of HIV research by publishing original research, full-length/mini review articles and guest edited thematic issues. The novel pioneering work in the basic and clinical fields on all areas of HIV research covers: virus replication and gene expression, HIV assembly, virus-cell interaction, viral pathogenesis, epidemiology and transmission, anti-retroviral therapy and adherence, drug discovery, the latest developments in HIV/AIDS vaccines and animal models, mechanisms and interactions with AIDS related diseases, social and public health issues related to HIV disease, and prevention of viral infection. Periodically, the journal invites guest editors to devote an issue on a particular area of HIV research of great interest that increases our understanding of the virus and its complex interaction with the host.
D. Podzamczer*, N. Rozas, P. Domingo, C. Miralles, E. Van den Eynde, A. Romero, E. Deig, H. Knobel, J. Pasquau, A. Antela, B. Clotet, P. Geijo, E. Rodríguez de Castro, M.A. Casado, A. Muñoz, A. Casado and for the PRO-STR STUDY GROUP
Evangelia-Georgia Kostaki, Daniel Frampton, Dimitrios Paraskevis, Katerina Pantavou, Bridget Ferns, Jade Raffle, Paul Grant, Zisis Kozlakidis, Andria Hadjikou, Eirini Pavlitina, Leslie D. Williams, Angelos Hatzakis, Samuel R. Friedman, Eleni Nastouli and Georgios K. Nikolopoulos*
Dr. Yuntao Wu is a professor at the National Center for Biodefense and Infectious Diseases, George Mason University, Manassas, Virginia, USA. He received his Ph.D. from the Department of Microbiology and Immunology at Queen’s University, Kingston, Ontario, Canada, and his postdoctoral training at NIH, Bethesda, Maryland, USA. Dr. Wu studies HIV infection of blood CD4 T cells and macrophages (Science, 2001, 293:1503-6), mainly focusing on the role of chemokine- and HIV-1-mediated G-protein signaling and actin dynamics in HIV infection and pathogenesis (Cell, 2008, 134,782; Science Advances, 2019, 5:1,aat7911). Currently, his lab also studies the mechanisms of PSGL-1 restriction of HIV infection (Nature Microbiology, 2019, 4:813-25).