<![CDATA[Pneumocephalus]]> https://benthamscience.com RSS Feed for Disease Wise Article | BenthamScience EurekaSelect (+http://eurekaselect.com) Mon, 22 Jul 2024 05:32:38 +0000 <![CDATA[Pneumocephalus]]> https://benthamscience.com https://benthamscience.com <![CDATA[Cellular, Molecular and Non-Pharmacological Therapeutic Advances for the Treatment of Parkinson's Disease: Separating Hope from Hype]]>https://benthamscience.comarticle/92923 <![CDATA[Recent Advances in Pathophysiology of Traumatic Brain Injury]]>https://benthamscience.comarticle/84044

Methods: Initial damage force results in Primary brain injury, causing tissue destruction and distortion in the early post-injury period. These secondary injuries from TBI cause changes in cell performance and dissemination of trauma via activities like free-radical generation, depolarization, and formation of edema, excitotoxicity, and disruption of blood brain barrier, calcium homeostasis, and intracranial hematoma. The expectation for developing effect in TBI sufferers is the best knowledge of these activities and enhancement of remedies that restrict secondary brain damage.

Results: The focal point of this study is on knowing the complex outburst of secondary impairments and studying the pathophysiology of TBI which provides alternative treatment benefits.

Conclusion: While injured persons demonstrate dissimilar levels of harm and every case is novel with specific recovery profiles, this article strengthens the recent pathophysiological sight of TBI mainly attention on oxidative stress, excitotoxicity, cerebral oxygenation and cerebral blood flow (CBF), development of edema, and inflammatory activities. For initial research acknowledgment of these recurring factors could permit clarification of possible beneficial targets.]]> <![CDATA[ Intraoperative Magnetic Resonance Imaging]]>https://benthamscience.comarticle/17553 <![CDATA[ Gene Therapy for Pituitary Tumors]]>https://benthamscience.comarticle/6319